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Lori Lebson Phones & Addresses

  • 78 Walnut Hill Dr, Scituate, MA 02066 (443) 352-8308
  • Tampa, FL
  • s
  • 816 Shadebrush Rdg, West Chester, PA 19382
  • 7101 Travertine Dr #407, Baltimore, MD 21209
  • Zephyrhills, FL
  • Melbourne, FL
  • Williamsburg, VA
  • 816 Shadebrush Rdg, West Chester, PA 19382 (443) 352-8308

Resumes

Resumes

Lori Lebson Photo 1

Head, Us Medical Affairs

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Location:
78 Walnut Hill Dr, Scituate, MA 02066
Industry:
Pharmaceuticals
Work:
Teva Pharmaceuticals Aug 2016 - May 2018
Associate Director, Medical Affairs, Neurology and Psychiatry

Emd Serono, Inc. Aug 2016 - May 2018
Medical Director, Neurology and Immunology

Teva Pharmaceuticals Oct 2012 - Aug 2016
Senior Medical Science Liaison at Teva Neurology, Multiple Sclerosis

Johns Hopkins School of Medicine Mar 2009 - Nov 2012
National Ms Society Post Doctoral Fellow

University of South Florida Aug 2004 - Nov 2008
Doctoral Student
Education:
University of South Florida 2002 - 2004
Bachelors, Bachelor of Science
William & Mary 2000 - 2002
Satellite High School 1997 - 2000
Skills:
Western Blotting
Literature Reviews
Immunohistochemistry
Qualitative Research
Confocal Microscopy
Microscopy
Molecular Biology
Pcr
Cell Culture
Research Design
Spss
Transfection
Statistics
Molecular Cloning
Neuroscience
Pharmaceutical Industry
Clinical Trials
Clinical Research
Immunology
Medical Affairs
Life Sciences
Neurology
Lori Lebson Photo 2

Lori Lebson

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Publications

Us Patents

Monocytes As A Gene Delivery Vector For Secreted Proteins To Treat Alzheimer's Disease

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US Patent:
8518391, Aug 27, 2013
Filed:
Nov 2, 2009
Appl. No.:
12/610815
Inventors:
Dave Morgan - Clearwater FL, US
Siddharth G. Kamath - Tampa FL, US
Lori Lebson - Baltimore MD, US
Assignee:
University of South Florida - Tampa FL
International Classification:
A61K 48/00
C12N 5/078
US Classification:
424 9321, 424 9371
Abstract:
Disclosed is a method for the treatment of a neurodegenerative disorder by isolating a plurality of monocytic cells and introducing a vector containing a gene that expresses a protease capable of degrading amyloid peptide into the isolated monocytic cells. The modified cells are then administered to the patient. Preferably the introduced gene is selected from the group consisting of neprilysin, insulin degrading enzyme and endothelin converting enzyme. The protease capable of degrading amyloid peptide is secreted from the monocytic cells into the extracellular space thereof. According to one variant, the gene that expresses a protease capable of degrading amyloid peptide is the NEP gene with a deletion in the membrane binding domain and/or an appended signal peptide to drive secretion of the modified gene product. This invention contemplates the use of heterologous and autologos (the monocytic cells are obtained from the patient) transplants.
Lori Ann Lebson from Scituate, MA, age ~43 Get Report